Genome Integrity Discussion Group
Monday, June 4, 2018, 1:30 PM - 5:30 PM
The New York Academy of Sciences, 7 World Trade Center, 250 Greenwich St Fl 40, New York, USA
The New York Academy of Sciences
The greater New York metropolitan area is unparalleled in the concentration of world leading research on chromosome biology and function, as well as for research at the interface between chromosome integrity and the dynamics of malignancy. The Genome Integrity Discussion Group capitalizes on this concentration of excellence, providing a forum for interaction between basic- and clinically-oriented research groups working in these fields. These meetings facilitate synergy between labs, and provide a context in which previously unappreciated complementarities can be revealed.
In that spirit, the talks cover a broad range of areas including the DNA damage response and cancer predisposition, DNA replication, transcription, chromatin modification, recombination, cell cycle control, telomeres, chromosome segregation, epigenetic states, as well as the emergence of new technologies relevant to research in genome integrity. Although a primary focus is upon basic mechanisms and processes, these areas are pertinent to cancer and myriad human disease states.
Call for Student/Postdoc Presentation Abstracts: Deadline May 4, 2018
The year-end meeting consists of a scientific symposium including a keynote presentation and four early career investigator short talks selected from abstracts from 1:30 to 4:30 PM, followed by a poster session and networking reception from 4:30 to 6:00 PM. Please submit abstracts in CSHL format with file name NYAS.name.doc via email to Professor Keeney at email@example.com by May 4, 2018, for consideration.
Additional Support Provided by
June 04, 2018
Keynote Address: Timely Resolution Of DNA Secondary Structures Across The Genome
Coffee Break and Poster Set-up
Distinct Modes of DNA Polymerase α Recruitment for Leading- and Lagging-Strand Initiation have Implications for Genome Maintenance
Xrs2 and Tel1 Independently Contribute to Mre11 and Rad50-mediated DNA Tethering and Replisome Stability
ATR Undergoes Rapid Exchange at Sites of DNA damage in a Manner Dependent on its Kinase Activity
Discovery of a Novel Player in Meiotic Recombination in Mouse